日誌
香蕉的鉀離子可以降血壓？
Increased phosphorylation and thus activation of NCC allows for more Na+ reabsorption, consequently raising blood pressure.
增加磷酸化並因此激活 NCC 允許更多的 Na+(鈉離子)重吸收，從而提高血壓。
High extracellular [K+] is believed to activate specific phosphatases to dephosphorylate NCC, thereby reducing its activity.
高細胞外 [K+](鉀離子)被認為可以激活特定的磷酸酶，使 NCC 去磷酸化，從而降低其活性。
A high dietary potassium (K+) intake causes a rapid dephosphorylation, and hence inactivation, of the thiazide‐sensitive NaCl cotransporter (NCC)
高膳食鉀 (K+) 攝入量會導致噻嗪類敏感的 NaCl 協同轉運蛋白 (NCC) 快速去磷酸化，從而失活
Increased dietary K+ reduces blood pressure;
增加膳食 K+(鉀離子)可降低血壓；
Increasing potassium intake can help decrease your blood pressure if you have high blood pressure. By lowering blood pressure, increasing potassium intake can also reduce your risk for heart disease and stroke. In contrast, consuming too much sodium can raise your blood pressure.
如果您患有高血壓，增加鉀的攝入量有助於降低血壓。 通過降低血壓，增加鉀的攝入量也可以降低患心臟病和中風的風險。相反，攝入過多的鈉會升高血壓。
Potassium relaxes the walls of the blood vessels, lowering blood pressure and protecting against muscle cramping.
鉀可以放鬆血管壁，降低血壓並防止肌肉痙攣。
These data show that dietary potassium deficiency increases blood pressure largely by stimulating salt reabsorption along the distal nephron.
這些數據表明，膳食鉀缺乏主要通過刺激遠端腎單位對鹽分的重吸收來增加血壓。
Dietary supplementation with potassium can lower BP in normal and hypertensive patients.
膳食補充鉀可以降低正常人和高血壓患者的血壓。
